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These findings, with numerous related details, later presented in the text, are surprising, which requires further studies and attempts of understanding.ĭata on lesion mechanisms in post-traumatic myelin conditions are presented in various papers in the literature – including older ones, written by one of the authors (and contributors) of this article – with details and some new elements, as the research progresses in this area (some minimal general references to this subject are also found in a recent article of the first author of this article).
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AIS mean sensitive scores differ between the two lots, and also in favor of the study, but statistically significant only at discharge (p=0.048) the difference at admission is not significant (p=0.51) – possibly because of alcoholic-nutritional polyneuropathy. The American Spinal Injury Association Impairment Scale (AIS) mean motor scores differ significantly (tests: Mann-Whitney and t) between the control and study group in favor of the second, both at admission (p<0.001) and at discharge (p<0.001). Data processing has been achieved with SPSS 24. The applicative component of the article highlights statistical data resulted from a retrospective study regarding the specialized casuistry from the Neuromuscular Recovery Clinic of the “Bagdasar Arseni” Emergency Clinical Hospital, following the comparative analysis of two groups of patients with recent SCI: non-consumers – the control group (n=780) – and chronic ethanol consumers – the study group (n=225) – with the addition of a prospective pilot component. The subject is not frequently approached – according to a literature review with systematic elements, which we have done before – thus constituting a niche that deserves to be further explored.
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We present a synthesis of related data on lesion mechanisms in post-traumatic myelogenous disorders, namely some of the alcohols and their actions on the nervous system, with details on the influences exerted, in such afflictions, by the chronic consumption of ethanol. We found differences related to the neuro-functional deficiency and clinical progress, among non-consumers and chronic consumers of ethanol, with recent traumatic spinal cord injury (SCI).
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